Sequelae

Takotsubo syndrome (TTS) is caused by catecholamine surge, which is also observed in COVID-19 disease due to the cytokine storm.

The current coronavirus disease 2019 (COVID-19) presents an ongoing medical challenge, as it involves multiple organs, including the cardiovascular system. Takotsubo syndrome (TTS) has been described in the context of COVID-19 in 2 different scenarios: as a direct complication of the infection, and as an indirect outcome secondary to the psychological burden of quarantine and social isolation (ie, stress-induced cardiomyopathy). // 25.5.2021

The novel COVID-19 has had an unprecedented and devastating spread internationally. COVID-19 infection can lead to a number of cardiovascular sequelae, including heart failure, which may portend worse clinical outcomes. //

Nature Reviews Cardiology - In this Perspectives article, the authors highlight what is known about cardiovascular sequelae in survivors of COVID-19 and discuss important questions that need to be... // 22.10.2021

The SARS-CoV-2 infection determines the COVID-19 syndrome characterized, in the worst cases, by severe respiratory distress, pulmonary and cardiac fibrosis, inflammatory cytokine release, and immunosuppression. // 7.7.2021

A new study reported findings of semen analysis from a prospective COVID-19 cohort study. // 15.10.2021

Background Erectile dysfunction (ED), as the hallmark of endothelial dysfunction, could be a short- or long-term complication of COVID-19. Additionally, being ED a clinical marker and predictor... // 20.3.2021

Tuoreen tutkimuksen mukaan niin sanotut koronavarpaat ovat seurausta kehon immuunijärjestelmän tiettyjen osien aktivoitumisesta. Ihotautilääkäri Nicolas Kluger kertoo MTV Uutisille, että samankaltainen on joissain tapauksissa iskenyt myös rokotteen ottamisen jälkeen. // 14.10.2021

What is the relationship between COVID-19 and the brain? A new review published in Tropical Biomedicine summarizes the key findings on how severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causes severe COVID-19 infection, disrupts brain activity and produces a range of long-term neurological disorders. // 10.10.2021

The cardiovascular complications of acute COVID-19 are well described; however, a comprehensive characterization of the post-acute cardiovascular manifestations of COVID-19 at one year has not been undertaken. // 5.10.2021

The virus could be completely changing the way the pancreas beta cells work, Dr. Shiubing Chen told Insider. // 6.10.2021

Japanilainen mies kärsi koronan jälkimainingeissa ennennäkemättömästä "levottoman peräaukon oireyhtymästä". // 3.10.2021

45% of surviving patients reported at least 1 post-infection symptom after recovery from hospitalization for COVID-19; the most common symptoms were fatigue, sweating, chest tightness, anxiety, and myalgia. // 29.9.2021

Data shows that 67% of people with mild or moderate COVID-19 infection develop long COVID, with symptoms that last more than 30 days after a positive test. // 5.8.2021

An interesting new study discusses the occurrence of peripheral nerve injuries in a subset of patients who survived severe COVID-19. // 29.9.2021

Results of two studies may explain why some people develop diabetes after catching the virus // 29.9.2021

It has been demonstrated in recent studies that there is a significant breadth of emerging autoreactivity associated with severe severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. More specifically, researchers from Emory University have identified a relaxation of peripheral tolerance within early antibody secretion cells that arise in individuals infected with SARS-CoV-2, as important drivers of those responses. // 28.9.2021

Findings, not yet peer-reviewed, show people infected with coronavirus had a loss of brain volume even when the disease was not severe enough to require hospitalization // 28.9.2021

https://www.reddit.com/r/HermanCainAward/comments/pvw1av/this_is_someone_i_know_with_his_threeyearold/?utm_source=share&utm_medium=web2x&context=3

Tutkimukseen osallistui 781 henkilöä, joista noin puolet oli sairastanut koronan. // 27.9.2021

Reduced brain volume in people who have experienced COVID-19 resembles brain changes typically seen in older adults. The implications of these findings are not yet clear. // 24.9.2021

// 9.7.2021

Increasing evidence suggests that infection with Sars-CoV-2 causes neurological deficits in a substantial proportion of affected patients. While these symptoms arise acutely during the course of infection, less is known about the possible long-term consequences for the brain. //4.6.2020

Since the initial reports of coronavirus disease 2019 (COVID-19) in China in late 2019, infections from severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) have spread rapidly, resulting in a global pandemic that has caused millions of deaths //21.11.2020

There is strong evidence for brain-related pathologies in COVID-19, some of which could be a consequence of viral neurotropism, or of neuroinflammation following viral infection. // 18.8.2021

Background Clinical investigations have argued for long-term neurological manifestations in both hospitalized and non-hospitalized COVID-19 patients. It is unclear whether long-term neurological symptoms and features depend on COVID-19 severity. / 14.3.2021

Coronavirus disease 2019 (COVID-19) is likely to have long-term mental health effects on individuals who have recovered from COVID-19. //2.3.2021

There is emerging evidence of long-term sequelae in a considerable proportion of COVID-19 patients after recovery and the spectrum and severity of suc…

Nature Medicine - A comprehensive review of the current literature on post-acute COVID-19/ 22.2.2021