SARS-CoV-2 damages cardiomyocyte mitochondria and implicates long COVID-associated cardiovascular manifestations
Post-acute sequelae of SARS-CoV-2 cardiovascular symptoms are associated with trace-level cytokines that affect cardiomyocyte function
STUDY: Trace inflammation markers found up to 18 months post-infection.
Persistent inflammation impacts heart cell function, highlighting potential long-term heart risks for those with ongoing symptoms.
SARS-CoV-2 Damages Cardiomyocytes Mitochondria and Implicates Long COVID-associated Cardiovascular Manifestations
After Covid infection rest for a few months. This paper reveals mitochondrial damage to cardiomyocytes— cells that allow heart to beat. This is likely a reason for increased sudden cardiac death during exercise post acute Covid.