CCL2-mediated endothelial injury drives cardiac dysfunction in long COVID
Ongoing cardiac dysfunction in Long Covid is, in part, caused by upregulation of CCL2 by inflamed endothelial cells(thrombotic vascular tissue).
2025, THE GROWING SARSCOV2 REINFECTION ERA! 🔥Let me summarise, with today’s science, how reinfections can worsen your Cardiovascular outcomes and mortgage your future health: ➡️Cumulative heart damage: Reinfections can worsen myocardial injury or increase the risk of… pic.twitter.com/neUQElrZhu— Harry Spoelstra (@HarrySpoelstra) February 15, 2025
“Let me summarise, with today’s science, how reinfections can worsen your Cardiovascular outcomes and mortgage your future health:”
Doctors Warn COVID-19 Could Be a Hidden Trigger for Heart Attacks Long After Recovery
“A new study reveals that COVID-19 fuels dangerous plaque buildup in the arteries, making heart attacks and strokes more likely. Even after recovery, the risk remains high, stressing the importance of ongoing heart health monitoring.”
A new study reveals that COVID-19 fuels dangerous plaque buildup in the arteries, making heart attacks and strokes more likely. Even after recovery, the risk remains high, stressing the importance of ongoing heart health monitoring.
How the virus behind COVID-19 can harm your blood vessels and your heart
“How the virus behind COVID-19 can harm your blood vessels and your heart”
How the virus behind COVID-19 can harm your blood vessels and your heart
Even mild COVID raises the chance of heart attack and stroke. What to know about the risks ahead
“Between 30 days and a year after recovery from COVID, survivors were 52% more likely to have a stroke, 63% more likely to have a heart attack, and 72% more likely to develop heart failure.”
COVID-19 in the Initiation and Progression of Atherosclerosis: Pathophysiology During and Beyond the Acute Phase
SARS-CoV-2 infection can markedly influence the initiation and progression of atherosclerotic lesions.
- Endothelial dysfunction, platelet activation, and persistent inflammation are potential drivers of increased atherosclerosis following COVID-19.
‘Prevalence of youth suffering heart attacks goes up post-Covid’
"The prevalence of young patients with heart attacks has increased post-Covid...even if the person feels that he has recovered, he may not realise that the virus causes some changes in the endothelium that can cause heart attack & stroke."