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73 s655
73 s655

“In conclusion, SARS-CoV-2 has a profound and multifaceted impact on cardiac tissue, mediated primarily through the Spike protein's interaction with the ACE2 receptor.”

“The virus not only causes direct damage to the heart by infecting cardiomyocytes but also triggers significant systemic effects that can exacerbate preexisting cardiovascular conditions.”

“The extensive expression of ACE2 in heart cells underpins the high susceptibility of cardiac tissues to SARS-CoV-2, leading to a range of complications from myocarditis to severe myocardial dysfunction.”

·biomed.cas.cz·
73 s655
Post-acute sequelae of SARS-CoV-2 cardiovascular symptoms are associated with trace-level cytokines that affect cardiomyocyte function
Post-acute sequelae of SARS-CoV-2 cardiovascular symptoms are associated with trace-level cytokines that affect cardiomyocyte function

STUDY: Trace inflammation markers found up to 18 months post-infection.

Persistent inflammation impacts heart cell function, highlighting potential long-term heart risks for those with ongoing symptoms.

·nature.com·
Post-acute sequelae of SARS-CoV-2 cardiovascular symptoms are associated with trace-level cytokines that affect cardiomyocyte function
SARS-CoV-2 Damages Cardiomyocytes Mitochondria and Implicates Long COVID-associated Cardiovascular Manifestations
SARS-CoV-2 Damages Cardiomyocytes Mitochondria and Implicates Long COVID-associated Cardiovascular Manifestations
After Covid infection rest for a few months. This paper reveals mitochondrial damage to cardiomyocytes— cells that allow heart to beat. This is likely a reason for increased sudden cardiac death during exercise post acute Covid.
·medrxiv.org·
SARS-CoV-2 Damages Cardiomyocytes Mitochondria and Implicates Long COVID-associated Cardiovascular Manifestations