Study finds that HDL from Long COVID patients promotes key steps in early atherosclerosis.
Monocytes migrate more and take up more lipids, suggesting dysfunctional HDL may contribute to heart disease risk in Long COVID.
Even with mild symptoms during the acute phase, COVID still causes damage that gets missed with standard diagnostics.
In this study, all 15 patients had blood flow irregularities in their hearts after a COVID infection with mild symptoms.
And that's just scratching the surface.
A study of 343 people with mild COVID-19 found subtle but significant heart function changes months later.
“In conclusion, SARS-CoV-2 has a profound and multifaceted impact on cardiac tissue, mediated primarily through the Spike protein's interaction with the ACE2 receptor.”
“The virus not only causes direct damage to the heart by infecting cardiomyocytes but also triggers significant systemic effects that can exacerbate preexisting cardiovascular conditions.”
“The extensive expression of ACE2 in heart cells underpins the high susceptibility of cardiac tissues to SARS-CoV-2, leading to a range of complications from myocarditis to severe myocardial dysfunction.”
Covid DOUBLES risk of heart attack and stroke long-term.
In severe Covid, it's 4-7 times the risk.
Podcast on long-term risk of heart attack and stroke following Covid infection by Cleveland Clinic Preventive Cardiology co-section head Dr. Stanley Hazen, MD.
“…one in five heart attack patients are now younger than 40…. the timing is simply too coincidental for Covid not to be involved. Studies have shown that, once in the body, the virus can cause the heart to become inflamed…Over time, in extreme cases, this can damage the organ to the point it becomes too weak to adequately pump enough blood to the rest of your body, causing heart failure…and severe Covid is much more likely to damage the heart.”
People with lingering heart symptoms post-COVID show trace cytokine levels detectable only with advanced nanotech.
These cytokines, like IL-1β and IL-6, may harm heart cells even at low levels.
STUDY: Trace inflammation markers found up to 18 months post-infection.
Persistent inflammation impacts heart cell function, highlighting potential long-term heart risks for those with ongoing symptoms.
COVID-19 significantly raises the risk of major adverse cardiac events, including myocardial infarction and stroke, especially in hospitalized patients.
The risk of thrombotic events is higher in individuals with non-O blood types.
“The risk of MACE (major adverse cardiac events)was elevated in COVID-19 cases at all levels of severity”
