"SARS-CoV-2 has been shown to infect the male reproductive tract.. Autopsy studies of COVID-19 patients have revealed testicular inflammation, leukocyte infiltration, and impaired Leydig cell function. SARS-CoV-2 may also fragment sperm DNA, reducing fertilization potential.. [In addition,] SARS-CoV-2 may affect the epididymis, a critical site for sperm maturation. Inflammation and oxidative stress in the epididymis can impair sperm motility and viability.."

“The present study demonstrated that SARS-CoV-2 significantly reduced ejaculate volume, sperm count, concentration, viability, normal morphology, and total and progressive motility. Furthermore, SARS-CoV-2 led to a reduction in circulating testosterone level, but a rise in oestrogen, prolactin, and luteinizing hormone levels. These findings were associated with a decline in testosterone/luteinizing hormone ratio. Conclusions The current study provides compelling evidence that SARS-CoV-2 may lower male fertility”