! 1 The Root Cause of Aging and Chronic Disease - Dr. Isaac Eliaz
Galectin-3 represents one of the fastest-growing fields of medical research, with over 3,000 published studies showing its far-reaching impacts on aging and chronic disease.
Last Days Overcomers is dedicated to preparing and empowering believers for the time leading up to the Day that Jesus comes back. We provide resources and conferences to help believers understand God’s purposes for the Last Days.
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Drugs only mask inflammation, they do not correct the source. Many pain meds also cause vitamin and mineral deficiencies. This side effect can prevent long term healing. For example, NSAIDS (non steroidal anti-inflammatory drugs) can cause folic acid and vitamin C deficiency. Both of these vitamins are crucial for the body to be able to repair damaged cartilage, joints, tendons, and ligaments.
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*These statements have not been evaluated by the Food and Drug Administration. This video is not intended to diagnose, treat, cure or prevent any disease. It is strictly intended for educational purposes only. Additionally, this information is not intended to replace the advice of your physician. Dr. Peter Osborne is one of the most sought after alternative and nutritional experts in the world. A Diplomate with the American Clinical Board of Nutrition, a graduate of Texas Chiropractic College, and a doctor of pastoral science, Dr. Osborne is one of the world’s leading authorities on gluten, nutrition, and natural health. He is the founder GlutenFreeSociety.org, one of the world's largest informational sites on gluten sensitivity. In addition, he is the author of the best selling book, No Grain No Pain, published by Touchstone (Simon & Schuster). His work has been featured by PBS, Netflix, Amazon, Fox, and many other nationally recognized outlets. For more information, visit us at https://www.glutenfreesociety.org/ or call 281-903-7527
Profiling neuroprotective potential of trehalose in animal m... Neural Regeneration Research
ehalose on its underlying mechanisms in animal models of selected neurodegenerative disorders (tau pathology, synucleinopathy, polyglutamine tract, and motor neuron diseases). All animal studies on neurodegenerative diseases treated with trehalose published in Medline (accessed via EBSCOhost) and Scopus were considered. Of the 2259 studies screened, 29 met the eligibility criteria. According to the SYstematic Review Center for Laboratory Animal Experiment (SYRCLE) risk of bias tool, we reported 22 out of 29 studies with a high risk of bias. The present findings support the purported role of trehalose in autophagic flux and protein refolding. This review identified several other lesser-known pathways, including modifying amyloid precursor protein processing, inhibition of reactive gliosis, the integrity of the blood-brain barrier, activation of growth factors, upregulation of the downstream antioxidant signaling pathway, and protection against mitochondrial defects. The absence of adverse events and improvements in the outcome parameters were observed in some studies, which supports the transition to human clinical trials. It is possible to conclude that trehalose exerts its neuroprotective effects through both direct and indirect pathways. However, heterogeneous methodologies and outcome measures across the studies rendered it impossible to derive a definitive conclusion. Translational studies on trehalose would need to clarify three important questions: 1) bioavailability with oral administration, 2) optimal time window to confer neuroprotective benefits, and 3) optimal dosage to confer neuroprotection....
Trehalose promotes functional recovery of keratinocytes under oxidative stress and wound healing via ATG5ATG7 - ScienceDirect
Wounds are in an stress state, which precludes healing. Trehalose is a stress metabolite that protects cells under stress. Here, we explored whether t…
The role of type 2 diabetes in the association between habitual glucosamine use and dementia a prospective cohort study Alzheimer's Research & Therapy Full Text
Background Growing evidence has showed an association between habitual glucosamine use and type 2 diabetes (T2D). However, the effect of habitual glucosamine use on risk of dementia remains poorly understood. Our study aimed to examine the association between glucosamine use and risk of dementia and further to identify the mediating role of T2D in the association. Methods A total of 495,942 participants from UK Biobank who completed a questionnaire on habitual glucosamine use were included at baseline (2006–2010) and then followed up for incidence of dementia until 2020. Cox proportional hazard regressions were performed to calculate hazard ratios (HRs) and 95% confidence intervals (CIs) for incident dementia. Markov multi-state models were used to explore the role of incidence of T2D during the follow-up in the association. Results Overall, 18.80% of the participants reported habitual use of glucosamine at baseline. A total of 6831 dementia events were recorded during a median follow-up of 11 years. In fully adjusted models, habitual glucosamine use was associated with a significantly lower risk of dementia (HR = 0.87, 95% CI: 0.82–0.93). Multi-state models showed that the association between glucosamine use and dementia was mediated by the incidence of T2D during the follow-up (HR of dementia without T2D: 0.92, 95% CI: 0.86–0.99; HR of post-T2D dementia: 0.79, 95% CI: 0.67–0.93). Conclusions Our findings reveal that habitual use of glucosamine supplement is associated with a lower risk of dementia, which might be explained by incidence of T2D.
How the Wrong Dietary Fat Can Wreck Your Health- Interview with Georgi Dinkov
In this interview, Georgi Dinkov, an expert on linoleic acid (LA), details some of the health hazards of this exceedingly common fat in the modern diet, and how to safely rid your body of it. Omega-6 linoleic acid (LA) is the most common fat in t…
Plant Based Kidneys; Michele is a registered dietitian / certified specialist in renal nutrition. Self-study program, friendly plant based kidney recipes, blog.
Episode 19: Role of Vitamin K2 in Kidney Disease
⏰ Timestamps ⏰
0:00 Introduction
0:45 What is Vitamin K and what is its role in kidney disease?
10:24 How do you get Vitamin K2 from your diet?
14:03 Why should you combine D3 with K2?
15:05 What dosage of K2 should you take?
16:30 Should you check your Vitamin K levels checked?
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The information provided on this channel is for educational purposes only, and does not substitute for professional medical advice. Please consult a medical professional or healthcare provider for medical advice, diagnoses, or treatment.
Ways and Means of Cellular Reconditioning for Kidney Regeneration - FullText - American Journal of Nephrology 2022, Vol. 53, No. 2-3 - Karger Publishers
Background: Mitochondrial, lysosomal, and peroxisomal dysfunction; defective autophagy; mitophagy; and pexophagy, as well as the loss of glycocalyx integrity are known contributors to initiation and progression of diverse kidney diseases. Those cellular organelles are tightly interactive in health, and during development of a disease, damage in one