Testing COVID

Is Boston University the new Wuhan? Researchers there have skipped the required NIH framework for dangerous pathogens to create a chimeric SARS virus strain on American soil. #GainOfFunction #BostonUniversity POSTED: October 24, 2022

... and also whether the virus exists. Briefly, the PCR test can work if it is done properly. It isn't being done properly anywhere. The virus does exist.

(NaturalHealth365) Strange research at Johns Hopkins University suggests PCR testing campaign could be used to vax the planet.

The emergence of the betacoronavirus, SARS-CoV-2 that causes COVID-19, represents a significant threat to global human health. Vaccine development is focused on the principal target of the humoral immune response, the spike (S) glycoprotein, that mediates ...

Background: SARS-CoV-2 that causes COVID-19 disease and led to the pandemic currently affecting the world has been broadly investigated. Different studies have been performed to understand the ...

(NaturalHealth365) Ohio Poison Control issues a national warning about at-home COVID-19 rapid antigen tests containing a toxic chemical.

The U.S. Food and Drug Administration (FDA) on March 1 said that three rapid COVID-19 tests shouldn't be ...

(NaturalHealth365) After two years of testing mania the CDC admits that the PCR tests used to drive pandemic fears are not reliable.

(NaturalHealth365) A union group is calling for COVID rapid tests to be pulled because the test swabs contain a toxic chemical.

The COVID-19 pandemic has brought us many harsh lessons. Importantly, it has shown us how easy it is to manufacture panic and control entire populations through deceptive means

COVID assumptions - the assumptions people make about COVID, how dangerous it is, how it spreads and what we need to do to stop it - are running rampant and far more wildly than the supposed virus

Whether you live in the USA or most of Europe, one thing remains true: Covid-19 testing regimes are being aggressively pushed onto populations

In medicine, we are taught not to guess when we can know, but that basic ethos for safe practice and the sharing of accurate information has not been applied to COVID-19

Hundreds of people across the United States have been complaining that they are receiving calls from clinics and medical offices claiming that they have confirmed positive for C0VlD but those people say they were never tested. Is this just an internet rumor or is it really happening? We're investigating. Check out our new free speech platform https://ISE.Media Invest in ISE Media: https://wefunder.com/ise.media ---------------- Support Truth in Media by visiting our sponsors: ISE.Media: https://ISE.Media Pulse Cellular: Use code "TRUTH" for 10% every plan for life. https://truthinmedia.com/phone Pure VPN: Military grade vpn protection. https://truthinmedia.com/vpn Brave Browser: Open source and built by a team of privacy focused, performance oriented pioneers of the web. https://truthinmedia.com/brave #benswann

Rationale Pre-clinical and autopsy studies have fueled the hypothesis that a dysregulated vascular endothelium might play a central role in the pathogenesis of ARDS and multi-organ failure in COVID-19. Objectives To comprehensively characterize and quantify microvascular alterations in patients with COVID-19. Methods Hospitalized adult patients with moderate-to-severe or critical COVID-19 (n = 23) were enrolled non-consecutively in this prospective, observational, cross-sectional, multi-center study. Fifteen healthy volunteers served as controls. All participants underwent intravital microscopy by sidestream dark field imaging to quantify vascular density, red blood cell velocity (VRBC), and glycocalyx dimensions (perfused boundary region, PBR) in sublingual microvessels. Circulating levels of endothelial and glycocalyx-associated markers were measured by multiplex proximity extension assay and enzyme-linked immunosorbent assay. Measurements and main results COVID-19 patients showed an up to 90% reduction in vascular density, almost exclusively limited to small capillaries (diameter 4–6 µm), and also significant reductions of VRBC. Especially, patients on mechanical ventilation showed severe glycocalyx damage as indicated by higher PBR values (i.e., thinner glycocalyx) and increased blood levels of shed glycocalyx constituents. Several markers of endothelial dysfunction were increased and correlated with disease severity in COVID-19. PBR (AUC 0.75, p = 0.01), ADAMTS13 (von Willebrand factor-cleaving protease; AUC 0.74, p = 0.02), and vascular endothelial growth factor A (VEGF-A; AUC 0.73, p = 0.04) showed the best discriminatory ability to predict 60-day in-hospital mortality. Conclusions Our data clearly show severe alterations of the microcirculation and the endothelial glycocalyx in patients with COVID-19. Future therapeutic approaches should consider the importance of systemic vascular involvement in COVID-19.