SARS-CoV-2 infection and viral fusogens cause neuronal and glial fusion that compromises neuronal activity
SARS-CoV-2 infection and viral fusogens cause neuronal and glial fusion that compromises neuronal activity
Neurologic Manifestations of Long COVID Disproportionately Affect Young and Middle‐Age Adults
“Neurologic Manifestations of Long COVID Disproportionately Affect Young and Middle-Age Adults”
Neurologic Manifestations of Long COVID Disproportionately Affect Young and Middle-Age Adults
SARS-CoV-2 and HSV-1 Induce Amyloid Aggregation in Human CSF Resulting in Drastic Soluble Protein Depletion
SARS-CoV-2 and HSV-1 induce amyloid aggregation, depleting soluble proteins.
Suggests a link between viral infections and neurodegenerative diseases via protein loss.
COVID and the brain: researchers zero in on how damage occurs
SARS-CoV-2 preferentially infects astrocytes, which support neuron function, potentially leading to symptoms like fatigue and confusion.
SARS-CoV-2 can also infect pericytes, leading to restricted blood flow and small-vessel strokes.
SARS-CoV-2 Invasion and Pathological Links to Prion Disease
Nearly half of those infected with SARS-CoV-2 experience long-term neurological problems, including brain fog and memory issues.
An infection may accelerate or exacerbate prion diseases like Creutzfeldt–Jakob disease, leading to rapid neurodegeneration.