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Three years after your initial covid infection, there were still *new* symptoms starting, mostly neurological or GI related. 'That mini stroke that you are having now could be related to that covid infection you had three years ago.' TY
Three years after your initial covid infection, there were still *new* symptoms starting, mostly neurological or GI related. 'That mini stroke that you are having now could be related to that covid infection you had three years ago.' TY
Three years after your initial covid infection, there were still new symptoms starting, mostly neurological or GI related. 'That mini stroke that you are having now could be related to that covid infection you had three years ago.'
·x.com·
Three years after your initial covid infection, there were still *new* symptoms starting, mostly neurological or GI related. 'That mini stroke that you are having now could be related to that covid infection you had three years ago.' TY
Long-term neurological and gastrointestinal sequelae of SARS-CoV-2, influenza, and other neurotropic infections with and without vaccination
Long-term neurological and gastrointestinal sequelae of SARS-CoV-2, influenza, and other neurotropic infections with and without vaccination
Long-term neurological and gastrointestinal sequelae of SARS-CoV-2, influenza, and other neurotropic infections with and without vaccination
Long-term neurological and gastrointestinal sequelae of SARS-CoV-2, influenza, and other neurotropic infections with and without vaccination
·medrxiv.org·
Long-term neurological and gastrointestinal sequelae of SARS-CoV-2, influenza, and other neurotropic infections with and without vaccination
“Multiple hit” model shows that COVID protein potentiates subsequent infection-related neuroinflammation and other physiological problems - PolyBio Research Foundation
“Multiple hit” model shows that COVID protein potentiates subsequent infection-related neuroinflammation and other physiological problems - PolyBio Research Foundation
University of Colorado study in rats demonstrates that SARS-CoV-2 protein activity drives longer-term vulnerability to another infection, augmenting physiological problems such as neuroinflammation, autonomic dysregulation, and hormonal imbalances
University of Colorado study in rats demonstrates that SARS-CoV-2 protein activity drives longer-term vulnerability to another infection, augmenting physiological problems such as neuroinflammation, autonomic dysregulation, and hormonal imbalances
·polybio.org·
“Multiple hit” model shows that COVID protein potentiates subsequent infection-related neuroinflammation and other physiological problems - PolyBio Research Foundation
SARS-CoV-2 infects neurons and induces neuroinflammation in a non-human primate model of COVID-19
SARS-CoV-2 infects neurons and induces neuroinflammation in a non-human primate model of COVID-19
“Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the etiologic agent of coronavirus disease 2019 (COVID-19), can induce a plethora of neurological complications in some patients. However, it is still under debate whether SARS-CoV-2 directly infects the brain or whether CNS sequelae result from systemic inflammatory responses triggered in the periphery. By using high-resolution microscopy, we investigated whether SARS-CoV-2 reaches the brain and how viral neurotropism can be modulated by aging in a non-human primate model of COVID-19.”
·cell.com·
SARS-CoV-2 infects neurons and induces neuroinflammation in a non-human primate model of COVID-19
2023
2023

SARS-CoV-2 Spike Protein Accumulation in the Skull-Meninges- Brain Axis: Potential Implications for Long-Term Neurological Complications in post-COVID-19 Zhouyi Rong1,2,15†, Hongcheng Mai1,2,15†, Saketh Kapoor1†, Victor G. Puelles3,4,13,14,

·biorxiv.org·
2023
Dumb and Dumber?
Dumb and Dumber?

Dumb and Dumber? IQ loss on every COVID-19 infection?

⁃Your doctor & surgeon ⁃Your pilot ⁃Your heads of state ⁃Driver in the car next to you ⁃Your brain 🧠?

Studies have provided substantial evidence that COVID-19 damages the brain, revealing a range of cognitive and neurological damage:

·x.com·
Dumb and Dumber?
Cognitive dysfunctions in the course of SARS‑CoV‑2 virus infection, including NeuroCOVID, frontal syndrome and cytokine storm (Review)
Cognitive dysfunctions in the course of SARS‑CoV‑2 virus infection, including NeuroCOVID, frontal syndrome and cytokine storm (Review)
𝘾𝙤𝙜𝙣𝙞𝙩𝙞𝙫𝙚 𝙙𝙮𝙨𝙛𝙪𝙣𝙘𝙩𝙞𝙤𝙣𝙨 𝙞𝙣 𝙩𝙝𝙚 𝙘𝙤𝙪𝙧𝙨𝙚 𝙤𝙛 𝙎𝘼𝙍𝙎‑𝘾𝙤𝙑‑2 𝙫𝙞𝙧𝙪𝙨 𝙞𝙣𝙛𝙚𝙘𝙩𝙞𝙤𝙣, 𝙞𝙣𝙘𝙡𝙪𝙙𝙞𝙣𝙜 𝙉𝙚𝙪𝙧𝙤𝘾𝙊𝙑𝙄𝘿, 𝙛𝙧𝙤𝙣𝙩𝙖𝙡 𝙨𝙮𝙣𝙙𝙧𝙤𝙢𝙚 𝙖𝙣𝙙 𝙘𝙮𝙩𝙤𝙠𝙞𝙣𝙚 𝙨𝙩𝙤𝙧𝙢
·spandidos-publications.com·
Cognitive dysfunctions in the course of SARS‑CoV‑2 virus infection, including NeuroCOVID, frontal syndrome and cytokine storm (Review)
Unraveling the enigma of long COVID: novel aspects in pathogenesis, diagnosis, and treatment protocols
Unraveling the enigma of long COVID: novel aspects in pathogenesis, diagnosis, and treatment protocols

“The nervous system, particularly the brain, can become infected by SARS-CoV-2 early in the course of COVID-19 via viral access through the cribriform plate located at the upper part of the nose.. this route is more ominous than previously thought. The pits of the cribriform plate and areas beneath the olfactory bulb including potential spaces such as the air sinuses can be a reservoir for not only viral persistence, but also its replication.. the upper part of the nose and the structures surrounding it appear to be ideal sites for access to the CNS (central nervous system).. This can cause a low-grade inflammation in the meninges and deeper tissues in the neuronal environment triggering neuroinflammation.. [This can cause] Symptoms such as brain fog, cognitive deficits, prolonged anosmia, memory deficits, and deposition of amyloid-like fibrils that cause premature aging in patients with long COVID.."

It's certainly ominous that SARS-CoV-2 can infect my brain instantly and silently.”

·link.springer.com·
Unraveling the enigma of long COVID: novel aspects in pathogenesis, diagnosis, and treatment protocols
Cell invasive amyloid assemblies from SARS-CoV-2 peptides can form multiple polymorphs with varying neurotoxicity
Cell invasive amyloid assemblies from SARS-CoV-2 peptides can form multiple polymorphs with varying neurotoxicity

𝘼𝙢𝙮𝙡𝙤𝙞𝙙 𝙖𝙜𝙜𝙧𝙚𝙜𝙖𝙩𝙚𝙨 𝙛𝙧𝙤𝙢 𝙎𝘼𝙍𝙎-𝘾𝙤𝙑-2 𝙥𝙚𝙥𝙩𝙞𝙙𝙚𝙨 𝙧𝙚𝙖𝙙𝙞𝙡𝙮 𝙚𝙣𝙩𝙚𝙧 𝙣𝙚𝙪𝙧𝙤𝙣𝙖𝙡 𝙘𝙚𝙡𝙡𝙨, 𝘢𝘯𝘥 𝘵𝘩𝘪𝘴 𝘤𝘢𝘶𝘴𝘦𝘴 𝘢 𝘳𝘦𝘥𝘶𝘤𝘵𝘪𝘰𝘯 𝘪𝘯 𝘵𝘩𝘦𝘪𝘳 𝘮𝘪𝘵𝘰𝘤𝘩𝘰𝘯𝘥𝘳𝘪𝘢𝘭 𝘳𝘦𝘴𝘱𝘪𝘳𝘢𝘵𝘪𝘰𝘯.

·biorxiv.org·
Cell invasive amyloid assemblies from SARS-CoV-2 peptides can form multiple polymorphs with varying neurotoxicity