University of Texas researchers found that Omicron BA.1 caused lasting neurological issues in hACE2 mice, linked to blood-brain barrier breakdown, clotting, and inflammation.
Viral RNA and spike protein lingered in the brain even without live virus.
University of Texas researchers found that Omicron BA.1 caused lasting neurological issues in hACE2 mice, linked to blood-brain barrier breakdown, clotting, and inflammation.
Viral RNA and spike protein lingered in the brain even without live virus.
Exploring the Persistence of the Spike Protein along the Skull-Meninges-Brain Axis and the Neurological Effects of COVID-19
This analysis is supported by numerous videos and illustrations.
Evidence that a COVID-19 infection can induce neurological sequelae.
The SARSCoV2 spike protein can persist in the brain—skull bone marrow and meninges—to induce neurologic damage
SARS-CoV-2 spike protein accumulates & persists in the body for years after infection, especially in the skull-meninges-brain axis, potentially driving long COVID. mRNA vaccines help but cannot stop it.
In mice, it caused inflammation, anxiety, and worsened brain injuries. Vaccines reduced but did not fully eliminate it.