"Patients suffering from COVID-19 have been found to exhibit dementia-related biomarkers.. The findings of this study suggest a potential link between mild-to-moderate SARS-CoV-2 infection and the initiation or acceleration of brain β-amyloid pathology, and hint at the possibility of an increased future incidence of Alzheimer’s disease subsequent to the COVID-19 pandemic."
SARS-CoV-2 affects Alzheimer’s disease
Plasma proteomic evidence for increased β-amyloid pathology after SARS-CoV-2 infection
UK study of 1,252 people finds SARS-CoV-2 may accelerate Alzheimer’s-related brain changes.
Infected individuals showed shifts in key biomarkers, with greater effects in hospitalized patients and those with hypertension.
When SARS-CoV-2 INFECTION can facilitate an ATTACK on our BRAIN, by PRIONS and AMYLOIDS.
“When SARS-CoV-2 INFECTION can facilitate an ATTACK on our BRAIN, by PRIONS and AMYLOIDS. (🧵mega-thread 8 studies)”
Amyloid-forming potential of Betacoronavirus including SARS-CoV-2
“Amyloid-forming potential of Betacoronavirus including SARS-CoV-2”
Amyloid-forming potential of Betacoronavirus including SARS-CoV-2
SARS-CoV-2 and HSV-1 Induce Amyloid Aggregation in Human CSF Resulting in Drastic Soluble Protein Depletion
SARS-CoV-2 and HSV-1 induce amyloid aggregation, depleting soluble proteins.
Suggests a link between viral infections and neurodegenerative diseases via protein loss.