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Well, shit. SARS-Cov-2 causes impairment of the cholesterol efflux pathway, leading to foam cell formation and prion disease. Just like with HIV due to the protein found at citation #33. Talk about a clusterfuck.
Well, shit. SARS-Cov-2 causes impairment of the cholesterol efflux pathway, leading to foam cell formation and prion disease. Just like with HIV due to the protein found at citation #33. Talk about a clusterfuck.
“Well, shit. SARS-Cov-2 causes impairment of the cholesterol efflux pathway, leading to foam cell formation and prion disease. Just like with HIV due to the protein found at citation #33. “
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Well, shit. SARS-Cov-2 causes impairment of the cholesterol efflux pathway, leading to foam cell formation and prion disease. Just like with HIV due to the protein found at citation #33. Talk about a clusterfuck.
If you’re not noticing the (obvious) cognitive impairment in practically everyone around you, it may be due to your own cognitive decline.
If you’re not noticing the (obvious) cognitive impairment in practically everyone around you, it may be due to your own cognitive decline.

“If you’re not noticing the (obvious) cognitive impairment in practically everyone around you, it may be due to your own cognitive decline.

Or denial is rearing its ugly head once again.

Perhaps it’s easier for me considering my background, but honestly, it’s so bloody palpable.”

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If you’re not noticing the (obvious) cognitive impairment in practically everyone around you, it may be due to your own cognitive decline.
Studies re Long Covid insist on stating the % of people who succumb to this sequelae. I’ve seen incident rates from 3.5% up to 38% (after 3rd infection).
Studies re Long Covid insist on stating the % of people who succumb to this sequelae. I’ve seen incident rates from 3.5% up to 38% (after 3rd infection).

“GreyBut what every single one of these studies fail to mention is that the reduction in grey matter occurs in 100% of individuals"

A thread with studies:

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Studies re Long Covid insist on stating the % of people who succumb to this sequelae. I’ve seen incident rates from 3.5% up to 38% (after 3rd infection).
Ugh. The more we learn about it, the uglier it looks.
Ugh. The more we learn about it, the uglier it looks.

“Ugh. The more we learn about it, the uglier it looks.

This, again, speaks to my point about tail risk in an emergent situation: it’s at least as bad as it looks. It may be worse.

We are badly underestimating the consequences of delayed risk. It’s a reckless approach. (1/)”

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Ugh. The more we learn about it, the uglier it looks.
Can I reassert that with SARS-CoV-2 induced brain damage, it won’t be a lessening of intellect that you’ll be seeing first, but behavioural changes such as disinhibition, emotional lability, risk taking & impulsivity.
Can I reassert that with SARS-CoV-2 induced brain damage, it won’t be a lessening of intellect that you’ll be seeing first, but behavioural changes such as disinhibition, emotional lability, risk taking & impulsivity.

“Can I reassert that with SARS-CoV-2 induced brain damage, it won’t be a lessening of intellect that you’ll be seeing first, but behavioural changes such as disinhibition, emotional lability, risk taking & impulsivity.

Frontal lobe damage impairs behavioural regulation.” Thread:

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Can I reassert that with SARS-CoV-2 induced brain damage, it won’t be a lessening of intellect that you’ll be seeing first, but behavioural changes such as disinhibition, emotional lability, risk taking & impulsivity.