Clinical and CSF single-cell profiling of post-COVID-19 cognitive impairment
we found that cognitive dysfunction is common; is not influenced by mood, fatigue, or sleepiness; and is correlated with MRI changes in very few people. In a subgroup that
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“Long COVID can injure the brain - and persistent autoimmunity could be a major driver. A study links persistent AT1 receptor autoantibodies (AT1-AA) with neuroaxonal injury and cognitive symptoms. Here’s what it means🧵”
Long COVID can injure the brain - and persistent autoimmunity could be a major driver.
A study links persistent AT1 receptor autoantibodies (AT1-AA) with neuroaxonal injury and cognitive symptoms.
Here’s what it means🧵
Frontiers | Serum angiotensin type-1 receptor autoantibodies and neurofilament light chain as markers of neuroaxonal damage in post-COVID patients
“Dysregulation of autoimmune responses and the presence of autoantibodies (AA), particularly those related to the renin-angiotensin system (RAS), have been implicated in the acute phase of COVID-19, and persistent dysregulation of brain RAS by RAS-related autoantibodies may also contribute to neurological symptoms of post-COVID.”
Dysregulation of autoimmune responses and the presence of autoantibodies (AA), particularly those related to the renin-angiotensin system (RAS), have been implicated in the acute phase of COVID-19, and persistent dysregulation of brain RAS by RAS-related autoantibodies may also contribute to neurological symptoms of post-COVID.
Frontiers | The impact of COVID-19 on accelerating of immunosenescence and brain aging
COVID-19 induces chronic inflammation, immunosenescence, and neuroinflammation.