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COVID-19’s immuno-pathology and cardiovascular diseases
Transcriptional reprogramming from innate immune functions to a pro-thrombotic signature by monocytes in COVID-19
Transcriptional reprogramming from innate immune functions to a pro-thrombotic signature by monocytes in COVID-19
Small NIH study reveals how immune response triggered by COVID-19 may
"Scientists found evidence that antibodies--proteins produced by the immune system in response to viruses & other invaders--are involved in an attack on cells lining the brain’s blood vessels..." Cross-referenced in thread on Covid brain damage.
Blood-clotting imbalance persists in Long COVID, research finds
In addition to blood clotting disorders, specific cells of the immune system were at abnormal levels long after Covid infection:
Dysregulation of Protein S in COVID-19
"Acquired Protein S deficiency commonly occurs in COVID-19, with variable contribution to thromboembolic occurrence... Acquired PS deficiency is a relatively common complication of infection with human immunodeficiency virus 1 (HIV-1), varicella, dengue, and SARS-CoV-2, all of which are associated with an increased risk of thrombosis. Acquired PS deficiency is a relatively common complication of infection with human immunodeficiency virus 1 (HIV-1), varicella, dengue, and SARS-CoV-2, all of which are associated with an increased risk of thrombosis."