COVID more severe, longer-lasting than other respiratory diseases.
The findings support the histopathological evidence of an exuberant immune reaction associated with SARS-CoV-2 infection.
This is from 2021. We never heard about it again.
“As part of a new study on hybrid immunity to the virus, the large, multi-institution research team led by The University of Texas at Austin discovered and isolated a broadly neutralizing plasma antibody, called SC27, from a single patient. Using technology developed over several years of research into antibody response, the team led by UT engineers and scientists obtained the exact molecular sequence of the antibody, opening the possibility of manufacturing it on a larger scale for future treatments.”
Your immune system is a factory 🧵
T-cells are the essential workers.
SARS-CoV-2 -19 is a hacker that doesn't just disrupt operations from the outside.
It infiltrates the system and systematically lays off the workers (T-cells).
CO-INFECTION is the NEXT TIME BOMB 💥
SARS-CoV-2 + MYCOBACTERIUM TUBERCULOSIS (Mtb), Mtb which was before the pandemic, the leading infectious cause of death worldwide due to a single infectious agent
“Hey folks, ONE QUARTER of the global population (1.7 BILLION) carries the bacteria that causes tuberculosis (TB), Mycobacterium tuberculosis, in a dormant state. All it takes to activate >>> lowered immune functioning >>> SARS2.
Get it now?”
exactly.
This is the #Leonardi_Effect
This is exactly what @NjbBari3 and I have been saying for 3 years.
A sustained CD 4 count of <500 will get you there. That's data from HIV positive people.
@dbdugger has the receipts. He too has been warning of this inevitably.
This is a direct result of immune suppression due to Covid infection. HIV does the same thing.
“As the Kansas and Missouri medical communities prepare for respiratory illness season, health officials grapple with an early COVID-19 infection spike and higher-than-normal tuberculosis infections in Wyandotte County.”
T cell exhaustion in #MECFS & #LongCovid from predominant sympathetic state & the T cells engage the sympathetic fibers & adrenergic signaling decreases their effector function. Sympathetic nerves suppress T-cell responses in infection and in cancer
"Molecular Trickery: How Covid Silently Sabotages the Human Immune System... Researchers have discovered that SARS-CoV-2 manipulates the human immune system by forcing cells to produce non-functional proteins, hindering the body’s antiviral defenses."
Study from California State University analyzed immune responses to 35 viruses using data from 219 datasets.
Findings show SARS-CoV-2 triggers a unique cytokine-chemokine profile, distinct from other viruses.
Excellent explanation of mechanism discussed in recent SarsCov2 causing AIDS paper.
“So, to recap: Researchers identified two genotypes of the SARS-CoV-2 nucleocapsid protein that are able to use CD147 on host lymphocytes to infect those cells, and explain how that could contribute to immune deficiency and viral persistence (by citing independent research).”
A grim reality for SARS-CoV-2 survivors
Severe and lasting damage to their immune systems
‘“Tuberculosis after recovering from COVID-19 is becoming more common, potentially leading to a TB outbreak in the post-COVID-19 era.’
In only 3 years we discovered this.
HIV, discovered in 1981? Took until the early 90s.
And yet, the @CDC_TB never tells people to avoid C0Vid.”
“Conclusion Tuberculosis after recovering from COVID-19 is becoming more common, potentially leading to a TB outbreak in the post-COVID-19 era. The immunosuppressive nature of the disease and its treatment modalities may contribute to post COVID-19 TB.”
T-Cell-Derived 3-Gene Signature Distinguishes SARS-CoV-2 from Common Respiratory Viruses
This is YET ANOTHER study showing SARS-CoV-2 adversely impacts the immune system.
Monocyte abnormalities (decreases) were found in BOTH symptomatic and asymptomatic SARS-CoV-2 infections. Because monocytes INCREASE in normal immune response, this suggests monocyte infection.
