“Findings emphasize that Covid-19’s myocardial pathology predominantly arises from fibrin thrombi within the coronary MICROVASCULAR network, especially in the abluminal spaces.
➡️This pathological process leads to myocardial fibre injury, necrosis, and ischemic changes( = fibrosis), contributing to both the acute symptoms and long-term sequelae (Long Covid).”
Uptick in post-acute Covid avascular necrosis.
Just like what happened in SARS1 survivors, which was well-documented but our government chose to ignore and pretend Covid is a cold.
Covid, and even asymptomatic covid is an endothelial disease.
Polish study Study on endothelial cells shows SARS-CoV-2 disrupts cell elasticity, causing vascular damage even without full replication.
“Clinical data indicate that COVID-19 causes cardiovascular complications, regardless of the severity of the disease. In this work, we have shown that SARS-CoV-2 infection causes vascular dysfunction due to the modification of endothelial cell elasticity.”
The vascular endothelium, which normally maintains blood flow and resists clotting and inflammation, can become overactive during COVID-19.
This overactivation can lead to multiorgan complications and long-term effects associated with Long Covid.
“Disrupted endothelial function underlies the multiorgan complications of COVID-19”
“‘I couldn't believe what I saw; these amyloid signals were everywhere. And I got back to the control sample and did the same, and I saw little bits, but not much.’
Pretorius had become one of the first in the world to discover that COVID-19 could induce widespread clotting in the blood of patients.”
“About 20% of my young patients are showing evidence of increased clotting as part of the baseline lab panels.
Never used to see this.”
“Navigating social distancing requirements that complicated lab work, Akassoglou and her collaborators conducted a series of experiments in mice to explore the pernicious role of the coronavirus’s spike protein.
They discovered that beyond serving as the virus’s “key” to enter cells, spike binds with a blood clotting factor called fibrinogen, creating structurally abnormal, inflammation-promoting clumps of fibrin — the insoluble material that forms the mesh-like structures essential for wound healing.
High levels of these abnormal clots not only push the body’s clotting system into overdrive, increasing clot formation and inflammation, but also suppress natural killer cells — the immune system’s virus-clearing soldiers.”
