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SARS-CoV-2 S-protein expression drives syncytia formation in endothelial cells
SARS-CoV-2 S-protein expression drives syncytia formation in endothelial cells
US researchers found that SARS-CoV-2 spike protein causes endothelial cells to fuse into syncytia, weakening blood vessel barriers and potentially worsening COVID-19 complications.
#syncytia#endothelial inflammation#platelet endothelial cell adhesion molecule-1#persistent endothelial dysfunction#Endothelium#vascular#spike protein
·nature.com·
SARS-CoV-2 S-protein expression drives syncytia formation in endothelial cells
Endothelial inflammation in COVID-19
Endothelial inflammation in COVID-19

The vascular endothelium, which normally maintains blood flow and resists clotting and inflammation, can become overactive during COVID-19.

This overactivation can lead to multiorgan complications and long-term effects associated with Long Covid.

“Disrupted endothelial function underlies the multiorgan complications of COVID-19”

Disrupted endothelial function underlies the multiorgan complications of COVID-19
#vascular#Endothelium#Inflammation#endothelial inflammation#Post-Acute Conditions#organ damage
·science.org·
Endothelial inflammation in COVID-19