“Findings emphasize that Covid-19’s myocardial pathology predominantly arises from fibrin thrombi within the coronary MICROVASCULAR network, especially in the abluminal spaces.

➡️This pathological process leads to myocardial fibre injury, necrosis, and ischemic changes( = fibrosis), contributing to both the acute symptoms and long-term sequelae (Long Covid).”

Covid, and even asymptomatic covid is an endothelial disease.

Polish study Study on endothelial cells shows SARS-CoV-2 disrupts cell elasticity, causing vascular damage even without full replication.

“Clinical data indicate that COVID-19 causes cardiovascular complications, regardless of the severity of the disease. In this work, we have shown that SARS-CoV-2 infection causes vascular dysfunction due to the modification of endothelial cell elasticity.”

The vascular endothelium, which normally maintains blood flow and resists clotting and inflammation, can become overactive during COVID-19.

This overactivation can lead to multiorgan complications and long-term effects associated with Long Covid.

“Disrupted endothelial function underlies the multiorgan complications of COVID-19”

“About 20% of my young patients are showing evidence of increased clotting as part of the baseline lab panels.

Never used to see this.”