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Rth2
Rth2
"Coagulopathy has been recognized as a complication of COVID-19, and the incidence of thrombotic events may be as high as 31% among critically ill patients with the disease.80, 81 There is a concern that hormone therapy may compound this thrombotic risk."
·onlinelibrary.wiley.com·
Rth2
Pmc7301099
Pmc7301099
"...exogenous estrogens may increase coagulating factors and the risk of thromboembolic events with a potential consequent increase in mortality."
·ncbi.nlm.nih.gov·
Pmc7301099
Proteomics of fibrin amyloid microclots in long COVID/post-acute sequelae of COVID-19 (PASC) shows many entrapped pro-inflammatory molecules that may also contribute to a failed fibrinolytic system - Cardiovascular Diabetology
Proteomics of fibrin amyloid microclots in long COVID/post-acute sequelae of COVID-19 (PASC) shows many entrapped pro-inflammatory molecules that may also contribute to a failed fibrinolytic system - Cardiovascular Diabetology
More evidence that LongCOVID symptoms are due to widespread endothelial damage and a failed fibrinolytic system. We again found numerous entrapped inflammatory molecules; and this time also antibodies in microclots
·cardiab.biomedcentral.com·
Proteomics of fibrin amyloid microclots in long COVID/post-acute sequelae of COVID-19 (PASC) shows many entrapped pro-inflammatory molecules that may also contribute to a failed fibrinolytic system - Cardiovascular Diabetology
What Is a DVT?
What Is a DVT?

"COVID-19 increases people's risk of dangerous blood clots and bleeding for months after infection, researchers say.

The new findings suggest that COVID-19 is an independent risk factor for deep vein thrombosis, pulmonary embolism and bleeding."

·webmd.com·
What Is a DVT?
Jeff Gilchrist on Twitter
Jeff Gilchrist on Twitter

Detailed analysis thread on Twitter: COVID-19: Increased risk of dying from infection and sudden death

This a long thread looks at several studies on death and neurovascular injury along with a whole bunch of sudden death examples of relatively young and healthy people found by @MeetJess . 🧵1/

·twitter.com·
Jeff Gilchrist on Twitter
Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 (PASC) is accompanied by increased levels of antiplasmin - Cardiovascular Diabetology
Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 (PASC) is accompanied by increased levels of antiplasmin - Cardiovascular Diabetology
"Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 (PASC) is accompanied by increased levels of antiplasmin"
·cardiab.biomedcentral.com·
Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 (PASC) is accompanied by increased levels of antiplasmin - Cardiovascular Diabetology
COVID-19 and the Brain
COVID-19 and the Brain

From 2020: "Vascular events such as strokes have been reported in COVID-19 patients. It is thought that SARS-CoV-2 causes a thickening of the blood, which leads to blood clots"

·pennneuroknow.com·
COVID-19 and the Brain
Dr Claire Taylor on Twitter
Dr Claire Taylor on Twitter

1/A reminder that #LongCovid patients have microclots. We are sending children to school with no mitigations and a 5% chance of a disease that damages you blood vessels.🩸 ALL 80 patients tested in this paper had moderate microclots. Green is microclots.

·twitter.com·
Dr Claire Taylor on Twitter
Long covid clot risk
Long covid clot risk
“COVID-19 triggers a massive release of biomolecules from the immune system…these molecules can gum up the blood and make it more likely to form clots…a new study is showing that people with long COVID have a higher risk of blood clots.”
·tristatehomepage.com·
Long covid clot risk
A central role for amyloid fibrin microclots in long COVID/PASC: origins and therapeutic implications
A central role for amyloid fibrin microclots in long COVID/PASC: origins and therapeutic implications
“The result, as is strongly manifested in platelet-poor plasma (PPP) of individuals with Long COVID, is extensive fibrin amyloid microclots that can persist, can entrap other proteins, and that may lead to the production of various autoantibodies.”
·portlandpress.com·
A central role for amyloid fibrin microclots in long COVID/PASC: origins and therapeutic implications