Covid and Viral Persistence

Covid and Viral Persistence

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Chris Turnbull on Twitter
Chris Turnbull on Twitter

Detailed thread on Twitter New study shows that those infected by Sars2/Covid have pockets of compartmentalized virus in organs across the body, including the heart and testis containing new variants implying virus is able to spawn new variants from ongoing infection ala HIV—180 unique variants found.

·twitter.com·
Chris Turnbull on Twitter
B. Carfree on Twitter
B. Carfree on Twitter

The CDC recommends condom use for weeks (?) after one is infected with SARS-2.

We know many people have persistent infections in GI, testes, brains, and more, so it wouldn't be any big surprise if LC can spread it sexually or if it restarted replicating in respiratory tissue.

·twitter.com·
B. Carfree on Twitter
Dr. Gustavo Aguirre-Chang on Twitter
Dr. Gustavo Aguirre-Chang on Twitter

We reaffirm that the main cause of LongCOVID is the Viral Persistence It should not be considered Post-Infectious,Sequelae There is an active infection that mainly affects the blood vessels and there are often co-infections Something similar happens in CFS

·twitter.com·
Dr. Gustavo Aguirre-Chang on Twitter
Chris Turnbull on Twitter
Chris Turnbull on Twitter

Detailed Twitter thread: New study solidifies the fact that Covid reverse trabscibes into human DNA similar to HIV causing persistent infection:

·twitter.com·
Chris Turnbull on Twitter
Asymptomatic SARS-COV-2 infection in children's tonsils
Asymptomatic SARS-COV-2 infection in children's tonsils
"Positive immunostaining in adenotonsillar tissue samples suggest lymphoid tissue can be a reservoir of SARS-CoV-2 and may play an important role in community dissemination...unclear for how long the tissue can sustain SARS-CoV-2 in a persistent infection"
·sciencedirect.com·
Asymptomatic SARS-COV-2 infection in children's tonsils
Long COVID: A proposed hypothesis-driven model of viral persistence for the pathophysiology of the syndrome - PubMed
Long COVID: A proposed hypothesis-driven model of viral persistence for the pathophysiology of the syndrome - PubMed
"In this report, we propose a model for the pathophysiology of the long COVID presentation based on increased proinflammatory cytokine production that results from the persistence of the SARS-CoV-2 virus or one of its molecular components."
·pubmed.ncbi.nlm.nih.gov·
Long COVID: A proposed hypothesis-driven model of viral persistence for the pathophysiology of the syndrome - PubMed
Jenna Luche-Thayer on Twitter
Jenna Luche-Thayer on Twitter

here is a long list of peer-reviewed publications by many real scientists that support COVID-19: Reactivation – Recurrence/Viral Persistence

·twitter.com·
Jenna Luche-Thayer on Twitter