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Mitochondrial dysfunction in acute and post-acute phases of COVID-19 and risk of non-communicable diseases
Mitochondrial dysfunction in acute and post-acute phases of COVID-19 and risk of non-communicable diseases

Up to 70% of SARS-CoV-2 infected have reduced organ function for 4 months or more after infection, which leads to an increased risk of developing chronic diseases in e.g. lungs, kidneys and the cardiovascular system.

“Even though the etiology of COVID-19 is complex and there are multiple factors involved, decreased organ function has been associated with mitochondrial dysfunction for both the acute phase and post-acute phase of COVID”

·nature.com·
Mitochondrial dysfunction in acute and post-acute phases of COVID-19 and risk of non-communicable diseases
Sluggish gas exchange in the lungs may be involved in long-COVID brain fog | CIDRAP
Sluggish gas exchange in the lungs may be involved in long-COVID brain fog | CIDRAP

“Lower rates of gas exchange in the lungs may contribute to impaired cognitive function ("brain fog") tied to long COVID, according to a small study to be presented at next week's Radiological Society of North America's (RSNA's) annual meeting in Chicago.

Pulmonary gas exchange is the movement of oxygen from the lungs to the bloodstream and carbon dioxide from the bloodstream to the lungs.”

·cidrap.umn.edu·
Sluggish gas exchange in the lungs may be involved in long-COVID brain fog | CIDRAP
Did I not get the memo?
Did I not get the memo?
“Why do emergency department reports and hospital discharge summaries not mention that my patient tested positive for SARS-CoV-2 (or had “a really bad cold” or “the worst flu ever”) 1 month before they presented to hospital with weakness, falls, confusion, dyspnea, heart failure, acute kidney injury, or non–ST-elevation myocardial infarction? Doesn’t that seem relevant? It does to me.”pos
·cmaj.ca·
Did I not get the memo?