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JCI - Long COVID is associated with lower percentages of mature, cytotoxic NK cell phenotypes
JCI - Long COVID is associated with lower percentages of mature, cytotoxic NK cell phenotypes

UCSF study of 35 people with Long COVID had fewer mature NK cells 4 months after infection.

Lower NK cell levels linked to more symptoms like fatigue and brain fog.

NK cells clear infected cells, which may explain lasting symptoms.

·jci.org·
JCI - Long COVID is associated with lower percentages of mature, cytotoxic NK cell phenotypes
HIV/AIDS and Long COVID are Very Similar
HIV/AIDS and Long COVID are Very Similar
“HIV/AIDS & COVID-19, particularly long COVID, share several significant similarities, especially in terms of viral persistence, T cell damage, immune system dysfunction, & activation of other pathogens. These parallels are important for understanding the long-term effects of both infections and their impact on the immune system.”
·notesforfriends.com·
HIV/AIDS and Long COVID are Very Similar
With a healthy immune system, one can usually reach ketosis in a matter of hours of fasting, an important part of cycling through energy that helps to regenerate immune cells. In Long Covid, this is not possible due to persistent gluconeogenesis, thus ongoing immune dysfunction.
With a healthy immune system, one can usually reach ketosis in a matter of hours of fasting, an important part of cycling through energy that helps to regenerate immune cells. In Long Covid, this is not possible due to persistent gluconeogenesis, thus ongoing immune dysfunction.
“With a healthy immune system, one can usually reach ketosis in a matter of hours of fasting, an important part of cycling through energy that helps to regenerate immune cells. In Long Covid, this is not possible due to persistent gluconeogenesis, thus ongoing immune dysfunction.”
·x.com·
With a healthy immune system, one can usually reach ketosis in a matter of hours of fasting, an important part of cycling through energy that helps to regenerate immune cells. In Long Covid, this is not possible due to persistent gluconeogenesis, thus ongoing immune dysfunction.
Towards an understanding of physical activity-induced post-exertional malaise: Insights into microvascular alterations and immunometabolic interactions in post-COVID condition and myalgic encephalomyelitis/chronic fatigue syndrome
Towards an understanding of physical activity-induced post-exertional malaise: Insights into microvascular alterations and immunometabolic interactions in post-COVID condition and myalgic encephalomyelitis/chronic fatigue syndrome
"Upon physical activity, affected patients exhibit reduced systemic oxygen extraction & oxidative phosphorylation capacity.. these are mediated by dysfunctions in mitochondria & microcirculation.. maintained by.. immune activation."
·link.springer.com·
Towards an understanding of physical activity-induced post-exertional malaise: Insights into microvascular alterations and immunometabolic interactions in post-COVID condition and myalgic encephalomyelitis/chronic fatigue syndrome