Cancer

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Welcome To Breast Cancer Conqueror - Breast Cancer Conqueror
Welcome To Breast Cancer Conqueror - Breast Cancer Conqueror
About Dr V Dr. V Speaks 7 Essentials Coaches Shop Coaching Programs Private Coaching Success Stories Blog Podcasts Recipes Retreats Conquer Together Sarasota Contact is ... Read More
·breastcancerconqueror.com·
Welcome To Breast Cancer Conqueror - Breast Cancer Conqueror
Ivermectin inhibits the growth of glioma cells by inducing cell cycle arrest and apoptosis in vitro and in vivo - PubMed
Ivermectin inhibits the growth of glioma cells by inducing cell cycle arrest and apoptosis in vitro and in vivo - PubMed
Glioma, the most predominant primary malignant brain tumor, remains uncured due to the absence of effective treatments. Hence, it is imperative to develop successful therapeutic agents. This study aimed to explore the antitumor effects and mechanisms of ivermectin (IVM) in glioma cells in vitro and …
·pubmed.ncbi.nlm.nih.gov·
Ivermectin inhibits the growth of glioma cells by inducing cell cycle arrest and apoptosis in vitro and in vivo - PubMed
Ivermectin, a potential anticancer drug derived from an antiparasitic drug - PMC
Ivermectin, a potential anticancer drug derived from an antiparasitic drug - PMC
Ivermectin has powerful antitumor effects, including the inhibition of proliferation, metastasis, and angiogenic activity, in a variety of cancer cells. This may be related to the regulation of multiple signaling pathways by ivermectin through PAK1 kinase. ...
·ncbi.nlm.nih.gov·
Ivermectin, a potential anticancer drug derived from an antiparasitic drug - PMC
Progress in Understanding the Molecular Mechanisms Underlying the Antitumour Effects of Ivermectin - PubMed
Progress in Understanding the Molecular Mechanisms Underlying the Antitumour Effects of Ivermectin - PubMed
Ivermectin, a dihydro derivative of avermectin (AVM), was introduced into the veterinary, agricultural and aquaculture markets for animal health in 1981. Ivermectin was soon adopted in 1987 as a human medicine that was originally used for the treatment of onchocerciasis, a parasitic infection. Since …
·pubmed.ncbi.nlm.nih.gov·
Progress in Understanding the Molecular Mechanisms Underlying the Antitumour Effects of Ivermectin - PubMed
NaturalCancerCures.org - HOME
NaturalCancerCures.org - HOME
​Many people who are diagnosed with cancer are told that they need to have surgery, chemotherapy or radiation treatments. They are often told that  "There is nothing else they can do."
·naturalcancercures.org·
NaturalCancerCures.org - HOME
Is There A Cure for Skin Cancer - Iodine and Chlorine Dioxide to the Rescue
Is There A Cure for Skin Cancer - Iodine and Chlorine Dioxide to the Rescue
“I had three hard, very heavy tumors, so big that they almost prevented me from breathing. Then, at night, while sleeping, I had blood that would get into my nose and choke me. After two weeks of chlorine dioxide treatments using CDS [protocols C and D], I felt that the tumors were drying up more... View Article
·drsircus.com·
Is There A Cure for Skin Cancer - Iodine and Chlorine Dioxide to the Rescue
Mitochondria Health, Cancer, Chlorine Dioxide & Supporting Protocol
Mitochondria Health, Cancer, Chlorine Dioxide & Supporting Protocol
Hitting the Bull’s-Eye in Metastatic Cancers is the name of an interesting research paper published in the journal Pharmaceuticals in 2015. Its abstract reads: “Tumor metastases that impede the function of vital organs are a major cause of cancer-related mortality. Mitochondrial oxidative stress induced by hypoxia, low nutrient levels, or other stresses, such as genotoxic... View Article
·drsircus.com·
Mitochondria Health, Cancer, Chlorine Dioxide & Supporting Protocol
Six-month low level chlorine dioxide gas inhalation toxicity study with two-week recovery period in rats Journal of Occupational Medicine and Toxicology Full Text
Six-month low level chlorine dioxide gas inhalation toxicity study with two-week recovery period in rats Journal of Occupational Medicine and Toxicology Full Text
Background Chlorine dioxide (CD) gas has a potent antimicrobial activity at extremely low concentration and may serve as a new tool for infection control occupationally as well as publicly. However, it remains unknown whether the chronic exposure of CD gas concentration effective against microbes is safe. Therefore, long-term, low concentration CD gas inhalation toxicity was studied in rats as a six-month continuous whole-body exposure followed by a two-week recovery period, so as to prove that the CD gas exposed up to 0.1 ppm (volume ratio) is judged as safe on the basis of a battery of toxicological examinations. Methods CD gas at 0.05 ppm or 0.1 ppm for 24 hours/day and 7 days/week was exposed to rats for 6 months under an unrestrained condition with free access to chow and water in a chamber so as to simulate the ordinary lifestyle in human. The control animals were exposed to air only. During the study period, the body weight as well as the food and water consumptions were recorded. After the 6-month exposure and the 2-week recovery period, animals were sacrificed and a battery of toxicological examinations, including biochemistry, hematology, necropsy, organ weights and histopathology, were performed. Results Well regulated levels of CD gas were exposed throughout the chamber over the entire study period. No CD gas-related toxicity sign was observed during the whole study period. No significant difference was observed in body weight gain, food and water consumptions, and relative organ weight. In biochemistry and hematology examinations, changes did not appear to be related to CD gas toxicity. In necropsy and histopathology, no CD gas-related toxicity was observed even in expected target respiratory organs. Conclusions CD gas up to 0.1 ppm, exceeding the level effective against microbes, exposed to whole body in rats continuously for six months was not toxic, under a condition simulating the conventional lifestyle in human.
·occup-med.biomedcentral.com·
Six-month low level chlorine dioxide gas inhalation toxicity study with two-week recovery period in rats Journal of Occupational Medicine and Toxicology Full Text
'Vaccine-Induced Cancer' Chlorine Dioxide To The Rescue
'Vaccine-Induced Cancer' Chlorine Dioxide To The Rescue
We now have an inconceivable rise in cancer rates caused by COVID vaccines. Early in 2022, Senator Ron Johnson hosted a conference in Washington D.C. called “Second Opinion.” One of the speakers at this 5-hour recorded event last Monday was attorney Thomas Renz, who in the short time allotted to him, testified that three military... View Article
·drsircus.com·
'Vaccine-Induced Cancer' Chlorine Dioxide To The Rescue
Nutrient Deficiencies Linked to Breast Cancer - YouTube
Nutrient Deficiencies Linked to Breast Cancer - YouTube
Think you might be gluten sensitive? Take the quiz: https://www.glutenfreesociety.org/gluten-sensitivity-intolerance-self-test/ Due to the intestinal damage gluten can cause in susceptible individuals, digestion and nutrient absorption are hindered. This leads to nutrient deficiencies, including those linked to both cancer and gluten sensitivity. The mechanisms that link nutrient deficits and the progression of cancer are complex. Science has found that deficiencies in zinc, iron, vitamin B12, vitamin B6, and folate can damage your DNA, which can ultimately lead to cancer. It’s also worth noting that those eating a gluten-rich diet don’t usually get enough of the above nutrients to begin with. Because a gluten-rich diet is often filled with heavily processed foods that don’t contain adequate amounts of these nutrients. To connect with Dr. Osborne visit: On the web: https://drpeterosborne.com/ Facebook: https://www.facebook.com/DoctorPeterOsborne/ Pinterest: https://www.pinterest.com/docosborne/ Instagram: https://www.instagram.com/drosborne Twitter: https://twitter.com/glutenology *These statements have not been evaluated by the Food and Drug Administration. This video is not intended to diagnose, treat, cure or prevent any disease. It is strictly intended for educational purposes only. Additionally, this information is not intended to replace the advice of your physician. Dr. Peter Osborne is one of the most sought after alternative and nutritional experts in the world. A Diplomate with the American Clinical Board of Nutrition, a graduate of Texas Chiropractic College, and a doctor of pastoral science, Dr. Osborne is one of the world’s leading authorities on gluten, nutrition, and natural health. He is the founder GlutenFreeSociety.org, one of the world's largest informational sites on gluten sensitivity. In addition, he is the author of the best selling book, No Grain No Pain, published by Touchstone (Simon & Schuster). His work has been featured by PBS, Netflix, Amazon, Fox, and many other nationally recognized outlets. For more information, visit us at https://www.glutenfreesociety.org/ or call 281-903-7527
·youtube.com·
Nutrient Deficiencies Linked to Breast Cancer - YouTube
Researchers discover a dependency of glioblastoma on biotin distribution Further research hopes to find a successful combination of drugs that would cripple the growth of glioblastoma cells in patients
Researchers discover a dependency of glioblastoma on biotin distribution Further research hopes to find a successful combination of drugs that would cripple the growth of glioblastoma cells in patients
Researchers have discovered that the FDA-approved anti-fungal drug, sulconazole, exhibits anti-cancer properties towards glioblastoma cells. An in-depth investigation of this compound revealed that it competes with biotin (Vitamin H), an important co-factor for metabolic enzymes and modifier of histones, allowing it to inhibit the normal function of biotin-dependent metabolic enzymes and specific histone modification-associated gene expression.
·sciencedaily.com·
Researchers discover a dependency of glioblastoma on biotin distribution Further research hopes to find a successful combination of drugs that would cripple the growth of glioblastoma cells in patients
Naturopathic Oncology Care for Thoracic Cancers A Practice Survey
Naturopathic Oncology Care for Thoracic Cancers A Practice Survey
Background and Objectives: There is a lack of information on therapies recommended by naturopathic doctors (NDs) for lung and gastroesophageal cancer care. Study objectives were to: (1) identify the most common interventions considered for use by NDs; ...
·ncbi.nlm.nih.gov·
Naturopathic Oncology Care for Thoracic Cancers A Practice Survey
Selenomethionine or methylseleninic acid inhibits mutagenesis of a reporter gene in mouse bone marrow - PubMed
Selenomethionine or methylseleninic acid inhibits mutagenesis of a reporter gene in mouse bone marrow - PubMed
Recent laboratory and clinical studies have utilized selenium in the form of pure seleno-L-methionine (SeMet) in combination with DNA-damaging cancer chemotherapy drugs. In mice, the selenium protected bone marrow and other tissues from dose-limiting toxicity. In fact, because of the protection from …
·pubmed.ncbi.nlm.nih.gov·
Selenomethionine or methylseleninic acid inhibits mutagenesis of a reporter gene in mouse bone marrow - PubMed
Seleno-L-Methionine Modulation of Nucleotide Excision DNA Repair Relevant to Cancer Prevention and Chemotherapy - PubMed
Seleno-L-Methionine Modulation of Nucleotide Excision DNA Repair Relevant to Cancer Prevention and Chemotherapy - PubMed
Organic selenium compounds are known to prevent certain cancers although mechanisms may be complex. A widely-held view is that selenium compounds can induce apoptosis in cancer cells, or more precisely, in aberrant cells that are undergoing clonal evolution somewhere along the carcinogenesis process …
·pubmed.ncbi.nlm.nih.gov·
Seleno-L-Methionine Modulation of Nucleotide Excision DNA Repair Relevant to Cancer Prevention and Chemotherapy - PubMed
A Subpopulation of Stromal Cells Controls Cancer Cell Homing to the Bone Marrow Cancer Research
A Subpopulation of Stromal Cells Controls Cancer Cell Homing to the Bone Marrow Cancer Research
Breast and prostate cancer cells home to the bone marrow, where they presumably hijack the hematopoietic stem cell niche. We characterize here the elusive premetastatic niche by examining the role of mesenchymal stromal cells (MSC) in cancer cell homing. Decreasing the number of MSC pharmacologically enhanced cancer cell homing to the bone marrow in mice. In contrast, increasing the number of these MSCs by various interventions including G-CSF administration diminished cancer cell homing. The MSC subpopulation that correlated best with cancer cells expressed stem, endothelial, and pericytic cell markers, suggesting these cells represent an undifferentiated component of the niche with vascular commitment. In humans, a MSC subpopulation carrying markers for endothelial and pericytic cells was lower in the presence of cytokeratin+ cells in bone marrow. Taken together, our data show that a subpopulation of MSC with both endothelial and pericytic cell surface markers suppresses the homing of cancer cells to the bone marrow. Similar to the presence of cytokeratin+ cells in the bone marrow, this MSC subpopulation could prove useful in determining the risk of metastatic disease, and its manipulation might offer a new possibility for diminishing bone metastasis formation. Significance: These findings establish an inverse relationship between a subpopulation of mesenchymal stromal cells and cancer cells in the bone marrow. Cancer Res; 78(1); 129–42. ©2017 AACR .
·cancerres.aacrjournals.org·
A Subpopulation of Stromal Cells Controls Cancer Cell Homing to the Bone Marrow Cancer Research