Diabetes Type 1

Hello, dear friends! I'm Dr. Sean Hashmi, founder of SELF Principle, a community focused on the latest research in nutrition, longevity, health, wellness, and kidney disease. As a father, husband, and kidney disease specialist, my journey in medicine began with personal loss and my family's health struggles. These experiences ignited my passion to explore the healing power of nutrition and wellness. Here, we'll delve into scientific insights, engaging expert interviews, and real stories. More than just learning, this channel is about connecting, growing, and supporting one another on the path to better health. Your thoughts, challenges, and triumphs matter to me, so please SUBSCRIBE and join our VIP list by hitting the bell. Together, we'll embark on a journey toward understanding, compassion, and wellness. With heartfelt gratitude, Sean Hashmi, MD, MS, FASN

Learn about your kidneys, how to protect them, and what to eat when you have kidney disease! Dr. Sean Hashmi is a Board Certified Nephrologist and Michele Crosmer is a Registered Dietitian and Certified Specialist in Renal Nutrition. We hope to educate and empower you to take control of your health and make proactive lifestyle choices to protect kidney health! Thanks so much for subscribing to our channel! Sean Hashmi, MD, MS, FASN and Michele Crosmer, RD, CSR

By Richard J Johnson MD DrRichardJohnson.com Chronic kidney disease is not uncommon in the United States, with as many as 15 percent of all adults manifesting some decrease in kidney …

Fucoidin failed to protect the kidney from ischemic damage and was even nephrotoxic. It also stimulated the formation of iNOS RNA.

(NaturalHealth365) Did you know that starfruit contains a toxin that harms the kidneys and other internal organs, including the brain?

Raft-forming systems are designed to relieve reflux symptoms by forming a physical barrier on top of the stomach. The present study aimed to evaluate …

Insulin resistance, as a common pathological process of many metabolic diseases, including diabetes and obesity, has attracted much attention due to its relevant influencing factors. To date, studies have mainly focused on the shared mechanisms between mitochondrial stress and insulin resistance, and they are now being pursued as a very attractive therapeutic target due to their extensive involvement in many human clinical settings. In view of the complex pathogenesis of diabetes, natural drugs have become new players in diabetes prevention and treatment because of their wide targets and few side effects. In particular, plant phenolics have received attention because of their close relationship with oxidative stress. In this review, we briefly review the mechanisms by which mitochondrial stress leads to insulin resistance. Moreover, we list some cytokines and genes that have recently been found to play roles in mitochondrial stress and insulin resistance. Furthermore, we describe several natural drugs that are currently widely used and give a brief overview of their therapeutic mechanisms. Finally, we suggest possible ideas for future research related to the unique role that natural drugs play in the treatment of insulin resistance through the above targets.

LA SIN CENTRO STUDI SIN PRATICA CLINICA NEWS LA MISSIONE DELLA SOCIETÀ ITALIANA DI NEFROLOGIA Promuovere la ricerca e migliorare la cura delle malattie renali in Italia Scopri RIVISTE REGISTRI PROGETTI FORMAZIONE LA SIN CENTRO STUDI SIN PRATICA CLINICA NEWS […]

We evaluated Galectin-3 (Gal-3) as a potential early biomarker of acute kidney disease (AKI), and the effect of Gal-3 inhibition by modified citrus pe…

"Hit-and-run" treatment with senolytics, which in the case of D + Q have elimination half-lives

Conventional insulin concentration units (IU/mL or just U/mL) are bioefficacy based, whereas the Système International (SI) units (pmol/L) are mass based. In converting between these two different approaches, there are at least 2 well-accepted ...

After 20 years of treating patients and dealing with his own vein condition, Dr. Michael Ryan demonstrates his proven method of stopping a leg cramp in 15 se...

Check it out on AliExpress: https://s.click.aliexpress.com/e/_Ddxdwyr and be sure to come visit me at: https://www.DaveTheKayaker.com Also check out this blood glucose watch. So far I like it the most: https://youtu.be/4PzzQwqsWQw Subscribe and check out my "Fitness Tracker and Smartwatch Reviews" Playlist on YT. I like to bring you smartwatches that are new or different in some way, and this is the first one I've found with non-invasive blood glucose monitoring. #AliExpress #Smartwatch

What should we eat? I ask this question in the context of the incredible rates of obesity that we are now seeing, virtually worldwide. Today we will spend time with Dr. Jason Fung author of the book The Obesity Code. This is a landmark book that powerfully lays out why we are continuing to gain weight. It dispels the notion that dietary fat threatens to make us gain weight as well as the unfounded idea of calories in versus calories out as an explanation for why we gain weight. Dr. Fung clearly focuses on the importance of the hormone, insulin, in terms of obesity, and certainly in terms of diabetes as well. He outlines a very straightforward plan that is in perfect alignment with current science. This interview is one of the most fundamental as a relates to the science of weight loss and regaining control over our blood sugar. ==== 0:00 Intro 2:36 Why Dr. Fung Pursued Weight Loss Research 8:08 Problem with Insulin 15:15 Mechanisms of Insulin 23:09 Reconsidering Low Fat Diets & Calorie Counting 37:34 Satiety Hormones 40:10 Leptin & Weight Loss 43:59 Threat of Fructose 51:23 Conclusion ==== Dr. Jason Fung is a Canadian nephrologist and a world-leading expert on intermittent fasting and low carb, especially for treating people with type 2 diabetes. Dr. Fung graduated from the University of Toronto and completed his residency at the University of California, Los Angeles. He lives and works in Toronto, Canada. Here is Dr. Fung’s website: https://thefastingmethod.com/ https://www.dietdoctor.com/intermittent-fasting https://www.dietdoctor.com/low-carb https://www.dietdoctor.com/diabetes ___________________________ Instagram: https://www.instagram.com/davidperlmutter/ Website: https://www.drperlmutter.com/ Subscribe to our channel: https://www.youtube.com/channel/UCDRl_UAXxbHyOOjklnA0dxQ/?sub_confirmation=1

Know Labs, an emerging leader in non-invasive medical diagnostics, introduced the KnowU™, a new portable medical-grade glucose monitoring device that utilizes Bio-RFID™, Know Labs’ non-invasive diagnostic technology. Related Supersapiens Glucose Display Wearable Is Designed Specifically For Athletes The KnowU is a convenient, non-wearable, on-the-go alternative to fingersticks, which are used to check blood glucose levels, Know Labs introduced KnowU, a new portable medical-grade glucose monitoring device that utilizes Bio-RFID.

Learn all about new continuous glucose monitors (CGM) that won’t poke the skin.

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Highlights • In 2 different mouse models, AKI increased Gal-3 expression and induced cardiac dysfunction, cardiac and systemic inflammation, cardiac macrophage infiltration, and fibrosis. • Cardiac...

Background Galectin-3 (Gal-3) is a pleiotropic glycan-binding protein shown to be involved in sepsis and acute kidney injury (AKI). However, its role has never been elucidated in sepsis-associated AKI (S-AKI). We aimed to explore Gal-3’s role and its potential utility as a therapeutic target in S-AKI. Methods In 57 patients admitted to the intensive care unit (ICU) with sepsis, serum Gal-3 was examined as a predictor of ICU mortality and development of AKI. In a rat model of S-AKI induced by cecal ligation and puncture (CLP), 7-day mortality and serum Gal-3, Interleukin-6 (IL-6), and creatinine were examined at 2, 8, and 24 hours (h) post-CLP. Two experimental groups received the Gal-3 inhibitor modified citrus pectin (P-MCP) at 400 mg/kg/day and 1200 mg/kg/day, while the control group received water only (n = 18 in each group). Results Among 57 patients, 27 developed AKI and 8 died in the ICU. Serum Gal-3 was an independent predictor of AKI (OR = 1.2 [95% CI 1.1–1.4], p = 0.01) and ICU mortality (OR = 1.4 [95% CI 1.1–2.2], p = 0.04) before and after controlling for age, AKI, and acute physiology and chronic health evaluation (APACHE II) score. In the CLP rat experiment, serum Gal-3 peaked earlier than IL-6. Serum Gal-3 was significantly lower in both P-MCP groups compared to control at 2 h post-CLP (400 mg: p = 0.003; 1200 mg: p = 0.002), and IL-6 was significantly lower in both P-MCP groups at all time points with a maximum difference at 24 h post-CLP (400 mg: p = 0.015; 1200 mg: p = 0.02). In the Gal-3 inhibitor groups, 7-day mortality was significantly reduced from 61% in the control group to 28% (400 mg P-MCP: p = 0.03) and 22% (1200 mg P-MCP: p = 0.001). Rates of AKI per RIFLE criteria were significantly reduced from 89% in the control group to 44% in both P-MCP groups (400 mg: p = 0.007; 1200 mg: p = 0.007). Conclusions This translational study demonstrates the importance of Gal-3 in the pathogenesis of S-AKI, and its potential utility as a therapeutic target. Graphic abstract

We evaluated Galectin-3 (Gal-3) as a potential early biomarker of acute kidney disease (AKI), and the effect of Gal-3 inhibition by modified citrus pe…

Plant Based Kidneys; Michele is a registered dietitian / certified specialist in renal nutrition. Self-study program, friendly plant based kidney recipes, blog.